EATING CLEAN
Probiotics
Def.Live microorganisms that, when administered in adequate amounts, confer a health benefit on the host.
Recent evidence indicates that not only is our brain “aware” of our gut microbes, but these bacteria can influence our perception of the world and alter our behavior.
​
Fermented foods / goods
The microbiome (bacteria, viruses, fungus and parasites) that resides in your gut influence how we experience stress, the clarity of our thoughts, and quality of our mood. That's why we talk a lot about fermented foods in the Brain Food Clinic. Eating more kefir, kombucha, sauerkraut, kimchi, miso, and a little of sourdough is so healthy. Want to know more about fermented foods?
Complements
Healthy supplements:
- Magnesium
- Zinc
-Calcium
- Vitamins B
- Omega 3,6,7, : for membrane fluidity, nerve impulses.
-Proteins
- Fatty acids
-Antioxidants: Resveratrol, Polyphenols, vitamins A, E, C
-Amino acids
This promotes an ion balance - important for nerve impulses & ion exchanges K=, Na=, Ca, electrical potential and nerve conduction.
Prebiotics
Def. A selectively fermented ingredient that results in specific changes in the composition and/or activity of the gastrointestinal microbiota, thus conferring benefits upon host.
Plant-based Diet
Eat a diverse array of plant-based foods each day to benefit your digestive system, liver, gallbladder, and hormones. Your hormones will thank you for building a robust microbiome and reducing inflammation with nutrient-dense meals, full of color and deliciousness!
Individualized Medicine
.
Intestinal permeability
A healthy mucosa does not allow the passage of toxic substances (viruses, bacteria, allergens, toxins).
However, an imbalance of this occurs when taking anti-inflammatories, antibiotics, antacids, anxiolytics, antidepressants, stress, or an unbalanced diet. In other words, this barrier is broken and allows toxic substances to pass through, and micronutrients to be poorly absorbed. They also cause infections, intolerances, or allergies.
_edited.jpg)
Stress and your gut
Stress produces an excess of the hormone Cortisol, which depresses the immune system and increases glucose levels. This destroys the intestinal bacterial microbiota. The adrenaline secreted during periods of stress causes an alteration in digestive secretions, preventing the action of hydrochloric acid and digestive enzymes that suppress the correct digestion, fermentation and putrefaction of food in the intestinal lumen. This generates irritating substances for the mucosa and ends with an imbalance in the intestinal ecosystem. Favoring dysbiosis and the growth of Candida, which lead to intestinal permeability and with it a host of symptoms. Stress also causes the secretion of interferon gamma, disintegration of enterocytes and worsening of intestinal hyperpermeability.
Gluten on the intestine
You don't have to be celiac.
Gliadin, present in gluten, binds to a receptor (CXCR3) present in enterocytes (digestive tract cells), and triggers the release of Zonulin. This acts by opening the junctions of enterocytes. And causes the gap junctions to open and there is greater intestinal permeability. Gliadin is a low-digestibility protein, which also blocks the action of digestive proteases that help digest food.
Origin of intestinal permeability
It may be due to: intestinal dysbiosis, chronic candidiasis, intestinal infections, secretory IgA deficiency, allergenic foods, gluten and specific cereals, toxic products, stress, alcoholism, non-steroidal anti-inflammatory drugs, chemotherapy or radiotherapy.
Dysbiosis
It is one of the main causes of increased intestinal permeability.
Increased intestinal permeability has direct consequences on health. The passage of molecules means an increase in the entry of toxins into our system. This causes the saturation of the hepatic detoxification systems, increasing the possibility of having allergies, food sensitivities, and autoimmune processes.
Why allergies?
By allowing high molecular weight molecules to pass into the blood, this incorrect absorption creates a sensitization to the proteins of certain substances (foods), and IgG and/or IgE antibodies are formed. These immune complexes can act on the lungs, causing asthma or on the joints, causing arthritis. The following occur:
- IgE: food allergies
- IgG: food intolerances
And there is deficiency of IgA secretion.
Gluten intolerance
(NOT celiac)
Excessive gluten intake
Increases intestinal permeability
Increases the entry of Gliadin into the lamina propria
Increases the immune response
General damage to enterocytes
Causes of food intolerance
The causes of food intolerance are:
Absence of an enzyme necessary to fully digest a food. Lactose intolerance is a common example.
Irritable bowel syndrome . This chronic condition can cause cramping, constipation, and diarrhea.
For example, sulfites used to preserve fruits Sensitivity to food additives.
Dry foods, canned goods, and wine can trigger asthma attacks in sensitive people.
Recurrent stress or psychological factors . The reason is not fully known.
Celiac disease.
TO
-
to
Origin of intestinal permeability
-
Intestinal dysbiosis, chronic candidiasis, intestinal infections, secretory IgA deficiency, allergenic foods, gluten and specific cereals, toxic products, stress, alcoholism, anti-inflammatory drugs, non-steroidal drugs, chemotherapy or radiotherapy.
Types of lactose intolerance
There are four types of lactose intolerance:
- Primary lactase deficiency.
when people gradually lose the ability to digest milk due to a progressive loss of intestinal lactase synthesis. It varies greatly between 2 and 14 years of age.
- Secondary lactase deficiency.
These patients suffer from a decrease in the intestinal capacity to hydrolyze lactose due to damage to the enterocytes. Examples include gastroenteritis, celiac disease, intestinal resection and Crohn's disease.
- Development of lactase deficiency.
It occurs in newborns. In infants up to 2 years of age, the use of hydrolyzed formulas is recommended, which are accepted by 95% of children allergic to milk.
- Congenital lactase deficiency.
It consists of an autosomal recessive mutation in the LCT gene that encodes lactase located on the long arm of chromosome 2, which causes an enzyme with no or minimal activity.
to
-
-